MindMap Gallery local blood circulation disorder
Pathology and local blood circulation disorders are mainly caused by atherosclerosis, vascular stenosis, increased blood viscosity, or cerebral thrombosis, acute myocardial infarction, lower limb venous thrombosis and embolus loss caused by factors such as hypotension and slow blood flow. Pulmonary embolism, cerebral embolism, etc.
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This is a mind map about plant asexual reproduction, and its main contents include: concept, spore reproduction, vegetative reproduction, tissue culture, and buds. The summary is comprehensive and meticulous, suitable as review materials.
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local blood circulation disorder
Hyperemia (arterial congestion)
Cause
Arterioles dilate, blood flow accelerates, and the amount of perfusion into the circulation increases, resulting in congestion.
type
Physiological congestion
Pathological congestion
Inflammatory hyperemia (early stage of local inflammatory reaction)
Post-decompression hyperemia (caused by reflex expansion of arterioles in pressurized tissue)
Collateral hyperemia (open congestion of anastomotic branch arteries around ischemia)
Lesions and consequences
lesions
The volume increases, the color is bright red, the temperature rises and there is a pulsating sensation)
as a result of
Once the cause is eliminated, it can return to normal without any adverse effects.
Congestion (venous congestion)
Cause
venous compression
Local (the vein is compressed by various external reasons)
venous blood pooling
Local (gravity effect)
venous lumen obstruction
Local (venous thrombosis, formation of tumor thrombus by tumor cells that invade into veins)
heart failure
Systemic (left heart failure-pulmonary congestion, right heart failure-systemic organ congestion)
lesions
Swelling, local skin cyanosis or dark red, metabolism and temperature decrease)
ending
congestion edema
Congestive bleeding
Parenchymal cell atrophy, degeneration and even necrosis
Congestive sclerosis of organs
thrombus
forming conditions
cardiovascular endothelial cell damage
Anticoagulant effect of endothelial cells
Procoagulant effect of endothelial cells
changes in blood status
Increased blood coagulation type
Formation process and form
The process includes two basic processes: platelet aggregation and platelet coagulation.
Types and forms
White thrombus
mixed thrombus
red blood clot
hyaline thrombus
ending
dissolve, absorb
Mechanization, recanalization
Calcification
Effect on the body
block blood vessels
embolism
valvular heart disease
extensive bleeding
embolism
The way emboli travel
Emboli from systemic venous system and right heart
Can cause pulmonary embolism
Emboli from the left heart or arterial system
Commonly found in brain, lower limbs
Emboli from the mesenteric vein and other portal veins
Embolization of intrahepatic portal vein branches
cross emboli
The pressure in the right heart increases and the emboli pass through congenital atrial and ventricular septal defects to the left heart
retrograde embolism
Embolism type
Thromboembolism (the most common type of embolism)
Pulmonary embolism (95% of emboli come from the deep veins of the lower limbs)
Medium and small emboli often block small branches of the pulmonary artery
Large embolus embolizes the main trunk or large branches of the pulmonary artery
There are a large number of small emboli, which embolize extensively in most small branches of the pulmonary artery.
Systemic arterial embolism (80% of emboli come from the left heart)
The main sites are the lower limbs and brain, but the intestines, kidneys, and spleen can also be involved.
fat embolism
When long bone fractures, severe damage to subcutaneous adipose tissue, crush injuries or burns due to fatty liver, the adipose tissue ruptures to form lipid droplets, which enter the blood circulation through ruptured small veins.
Commonly found in organs such as lungs and brain
gas embolism
It is mostly caused by venous rupture and external air leaking out of the venous defect into the blood flow.
amniotic fluid embolism
Causes of sudden death
infarction
Conditions and causes of formation
blood vessel obstruction
blood vessel compression and occlusion
arterial spasm
Failure to establish effective collateral circulation
Local tissue tolerance to ischemia and systemic blood circulation status
type
anemic infarction
hemorrhagic infarction
septic infarction
Impact and outcome
Depends on the size and location of the infarct in the infarcted organ and whether there is bacterial infection (congestion, exudation-organized package-calcification)
consequences of embolism
Fresh thromboembolism is more harmful than old thromboembolism
Depends on the embolization site, local collateral circulation, and tissue tolerance to ischemia
The consequences of fat embolism depend on the location of the embolism and the number of fat droplets.
Gas embolism may cause dyspnea, cyanosis, and sudden death (decompression sickness and diver's syndrome)
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