Block vascular α1 receptors and relax blood vessels

Excites the heart, enhances myocardial contractility, accelerates heart rate, and increases cardiac output.

Treatment of peripheral vasospastic diseases

Norepinephrine drip leakage

Treatment of refractory congestive heart failure and acute myocardial infarction heart failure

Anti-shock

adrenal pheochromocytoma

For the identification, diagnosis and preparation before surgery of adrenal pheochromocytoma

drug-induced hypertension

Slow onset, strong and long-lasting effect

peripheral vasospasm

Anti-shock

Treating Pheochromocytoma

Treat benign prostatic hyperplasia

The heart rate slows down, myocardial contractility weakens, cardiac output decreases, myocardial oxygen consumption decreases, and blood pressure drops slightly.

Delays the conduction of the atria and atrioventricular nodes and prolongs the P-R interval of the electrocardiogram

Causes decreased blood flow to the liver, kidneys, and skeletal muscles

Inhibit lipolysis

Inhibit glycogenolysis

Suppress hyperthyroidism

Inhibit renin release

In addition to blocking, there is also partial activation

Some beta-blockers have

Block beta receptors in the ciliary body

Relevant to receptor upregulation

The main mechanism is to reduce the production of aqueous humor

No adverse reactions such as miosis and accommodation spasm

Treat renal insufficiency